Good Health Made SimpleBecause good health should be simple!

We held our first L.E.A.N. Essentials workshop last Saturday and the students LOVED it.  They were all health and nutrition-conscious coming in and so didn’t expect to learn as much from the class as they did.  They said they would definitely recommend these workshops to other parents they know.

If you are a parent of a child between the ages of 3-12, why not check them out for yourself?

The next 2-hour L.E.A.N. Essentials workshops will be held on:

Saturday, August 21 from 2:30-4:30pm
Tuesday, August 24 from 6:30-8:30pm
Tuesday, August 31 from 6:30-8:30pm

If you really want to go deep and learn about Traffic Light Eating, Label Reading, The Skinny on Fat, How to Develop Healthy Habits for the Entire Family and more, then you will want to attend the next 6-week L.E.A.N. Start workshop.  The 6-week series begins Tuesday, September 7 at 7:30pm.

Click HERE for more info on L.E.A.N. Start!

Finally, if you are a pregnant or nursing mom, you will absolutely want to attend one or all three of our L.E.A.N. Expectations workshops:

Prepare RIGHT Now on Saturday, August 28 from 2:30-4:30pm

Eat RIGHT Now on Saturday, September 4 from 2:30-4:30pm

Live RIGHT Now on Saturday, September 24 from 2:30-4:30pm

I wish you much health and happiness and I can’t wait to see you at a workshop very soon!  Take action now to secure your seat at a great discounted rate.

L.E.A.N. Coach Michele

Here’s an oldie but a goody that really hasn’t changed much:

Orlando, FL, October 6, 2008 – Results of a survey presented at the American College of Gastroenterology’s 73rd Annual Scientific Meeting in Orlando revealed that many parents do not accurately perceive their children as overweight or at risk for adulthood obesity. Obesity in the United States is often accompanied by an increased risk of gastrointestinal diseases and has emerged as a major health concern, particularly the issue of obesity among children and adolescents.
Researcher Rona L. Levy, Ph.D. and her colleagues at the University of Washington in Seattle and the University of Minnesota measured parental perceptions of their children’s current weight and perceived risk for developing obesity as an adult.
Forty-six parents of children ages 5 to 9 with a body mass index (BMI) in the 70th percentile or higher were recruited for the study. Child height and weight were measured during a routine pediatric clinic visit. Parents were mailed a series of questionnaires, which included questions on their perception of their child’s current weight, and whether they perceived that their child was at risk for developing obesity as an adult.
Dr. Levy and her research team found that even though all of the children had elevated BMI, less than 13 percent of the parents of overweight kids reported their child as currently overweight. Fewer than one-third perceived that their child’s risk for adult obesity was above average or very high.
“Clearly there is a significant misperception by parents of their child’s weight and risk for obesity,’ said Dr. Levy. “If we are going to address the growing epidemic of childhood obesity, parents’ description and awareness of their children’s overweight will have to be much more accurate,” said Dr. Levy.

About the American College of Gastroenterology
Founded in 1932, the American College of Gastroenterology (ACG) is an organization with an international membership of more than 10,000 individuals from 80 countries. The College is committed to serving the clinically oriented digestive disease specialist through its emphasis on scholarly practice, teaching and research. The mission of the College is to serve the evolving needs of physicians in the delivery of high quality, scientifically sound, humanistic, ethical, and cost-effective health care to gastroenterology patients.
The ACG is committed to providing accurate, unbiased and up-to-date health information. Visit the ACG Web site www.acg.gi.org to access educational resources for patients and their families spanning the broad range of digestive diseases and conditions – both common and not-so-common. Organized by disease, state and organ system, these educational materials, developed by ACG physician experts, are offered for the information and benefit of patients and the public.

What say you?  Do you think we’re so used to seeing overweight kids that it doesn’t register as unusual anymore?  See what I’m doing about it…

COLLEYVILLE, TX – Renowned pediatrician Dr. William Sears’ prestigious L.E.A.N. Start Program is being held this month for the first time at the Sunflower Shoppe Community Room in Colleyville, TX. CLICK HERE for complete class schedules and to register for classes.

The program allows parents and caregivers to “experience” nutrition and play, tasting a lot of healthy food and learning interactive games appropriate for children. It also demonstrates simple strategies to implement; including effective methods for prevention and intervention in families where nutrition related behaviors or childhood obesity are a concern.

A rising concern in recent years has been the increase in obesity among children and adolescents. For children between the years of 2 and 5 and adolescents between 12 and 19, the rate of obesity has doubled since the 1970s. For children between the ages of 6 and 11, the rate has tripled. Almost one third of all children today are overweight or obese.

The L.E.A.N. Start Program was developed by Dr. Sears, known as “America’s Pediatrician,” and Sean Foy, M.A., a leading exercise physiologist and nutrition expert. While most nutrition education programs target children, the L.E.A.N. Start Program is one of the first to be uniquely designed for parents and caregivers.

“To make a significant change in the health of our children, we must emphasize the role parents play in modeling healthy behavior, and educate parents on the benefits and principles of nutrition because they are responsible for grocery shopping, preparing meals, and shaping children’s tastes,” said Dr. Sears, who also is author of The Healthiest Kid in the Neighborhood. “There are a number of programs out there but they fail to impact the behavior of the parents. We’ve determined that in order to achieve real success, the education of parents is critical.”

The L.E.A.N. Start Program was first launched in Orange County, Calif. in 2007 and has trained more than 1,000 instructors. Dr. Sears created it after several years of trials and development in response to growing concerns over childhood obesity, nutrition deficit disorder and reported ineffectiveness of government-funded, child directed programs.

“Obesity isn’t the only problem our kids are facing, so we’ve designed a program that tackles a number of nutrition-related illnesses and issues,” said Foy. “The parents we see in our programs are motivated by wanting their children to be healthy, and ultimately we are seeing significant improvement in both the parents and children. Parents who completed the course had an average confidence level of 4.43 out of 5 for continuing the healthy behaviors.”

After evaluating pre and post-assessment clinical data dating from the program’s launch to July 2008, results showed that after completing the program, the number of participants who usually or always read nutritional labels before buying food increased 52 percent, while participants who considered nutrition when deciding which types of food to serve young children rose 19 percent to 77 percent. Additionally, the percentage of children of participating parents who ate three or more servings of fruits and vegetables increased 19 percent and 23 percent respectively.

Parents and caregivers who participated in the workshop also reported a substantial decrease in television and computer usage among young children and an increase in physical activity. Television and computer usage decreased by 28 and 37 percent respectively. The results are based on responses from 600 participants. Clinical data is still being collected, and research is ongoing.
For more information, or to register for the L.E.A.N. Start Program in Colleyville, please call 817.637.7569 or visit http://bit.ly/LEANschedule

About Dr. William Sears:
William Sears, M.D. practices pediatrics in partnership with his three pediatrician sons in San Clemente, Calif. Dr. Sears received his pediatric training at Harvard Medical School’s Children’s Hospital and The Hospital for Sick Children in Toronto and is an Associate Clinical Professor of Pediatrics at the University of California, Irvine School of Medicine. He is the author of more than 35 books on childcare and nutrition, and he has shared his expertise in more than 100 television programs including: Dr. Phil, The Doctors, Oprah, Dateline, 20/20 and Good Morning America where he is often referred to as “America’s Pediatrician.”

Contact
Michele Curtin
Good Health Made Simple
817.637.7569
LEANCoach.Michele (at) gmail.com

Check out the page I added with my Dr. Sears’ Certified L.E.A.N. Programs class schedule.  You can find it HERE.  These classes will sell out fast, so be sure to register for the class(es) of your choice today!  All classes are will be held in the Community Room at the Sunflower Shoppe in Colleyville, TX.

Be sure to check out the great events we’ve got planned for you this week in the DFW area.  You and your family will be blessed by attending!

Hope to see you there!

Click here to go to EVENTS PAGE

I was a little hesitant because it’s mainly unflavored with just a bit of real fruit at the bottom.  14 grams of protein, only 20 grams of carbs and 140 calories, so OK, I’ll give it a shot…

OMG!  It tastes amazing!  It is so thick and creamy and just yummy!

Chobani Yogurt is definitely worth trying today (and I don’t get paid a DIME for saying so)!

BTW, I found it at Costco..

Is it necessary to buy everything organic?  Ideal, yes, but necessary?  Probably not.

Generally, any fruit or vegetable where you eat the skin should be organic.  If you remove the skin, it safe to buy non-organic.

Here is a list  of the foods where it is best to buy organic, whenever possible.  Print this list and take it with you the next time you go grocery shopping:

FRUIT • Strawberries • Raspberries • Apples • Grapes • Raisins • Grape Juice • Peaches • Nectarines • Apricots • Pears • Cherries • Lemons • Limes • Bananas • Kiwis • Cantaloupes •  Tomatoes
VEGET ABLES • Potatoes • Carrots • Corn • Celery • Cucumbers • Spinach •    Green & Red Bell Peppers • Lettuce • Hot Peppers • Green Beans • Hard Winter Squash
OTHER • Rice • Oats • Milk • Baby Foods! • WILD Salmon • Anything that could be GMO – like Corn and Soy (GMO = Genetically Modified)
Here’s a great source to get organic produce delivered right to your door:  www.DeliciousOrganics.com

To your health,

Michele Curtin
“Mom on a Mission”

I just got done with a fun, informative, interactive, exhausting weekend of training in Denver CO.  I am awaiting my test results from the final exam and have full confidence that I will soon be certified as a Dr. Sears Certified LEAN Coach.  LEAN coaches facilitate a number of classes geared at stemming the tide of childhood obesity and all of the health challenges that ensue from that.

LEAN stands for Lifestyle, Exercise, Attitude and Nutrition.  Soon, I will be authorized to conduct classes teaching parents of kids ages 3-12 a set curriculum developed by Dr. Sears and his team.  I am so proud and thrilled to be associated in this way with America’s Pediatrician!

Stand by for more details as I get classes scheduled and start changing even more lives!

A new day is dawning for families in the DFW metroplex…

This list appears in Nancy Appleton’s book Lick The Sugar Habit

I recommend you print it out and post somewhere visible, like the fridge or pantry:
(See below for clinical documentation of each statement.)

1. Sugar can suppress the immune system.
2. Sugar upsets the mineral relationships in the body.
3. Sugar can cause hyperactivity, anxiety, difficulty concentrating, and crankiness in children.
4. Sugar can produce a significant rise in triglycerides.
5. Sugar contributes to the reduction in defense against bacterial infection (infectious diseases).
6. Sugar causes a loss of tissue elasticity and function, the more sugar you eat the more elasticity and function you lose.
7. Sugar reduces high density lipoproteins.
8. Sugar leads to chromium deficiency.
9. Sugar leads to cancer of the breast, ovaries, prostate and rectum.
10. Sugar can increase fasting levels of glucose.
11. Sugar causes copper deficiency.
12. Sugar interferes with absorption of calcium and magnesium.
13. Sugar can weaken eyesight.
14. Sugar raises the level of a neurotransmitters: dopamine, serotonin and norepinephrine.
15. Sugar can cause hypoglycemia.
16. Sugar can produce an acidic digestive tract.
17. Sugar can cause a rapid rise of adrenaline levels in children.
18. Sugar malabsorption is frequent in patients with functional bowel disease.
19. Sugar can cause premature aging.
20. Sugar can lead to alcoholism.
21. Sugar can cause tooth decay.
22. Sugar contributes to obesity.
23. High intake of sugar increases the risk of Crohn’s disease and ulcerative colitis.
24. Sugar can cause changes frequently found in persons with gastric or duodenal ulcers.
25. Sugar can cause arthritis.
26. Sugar can cause asthma.
27. Sugar greatly assists the uncontrolled growth of Candida Albicans (yeast infections).
28. Sugar can cause gallstones.
29. Sugar can cause heart disease.
30. Sugar can cause appendicitis.
31. Sugar can cause multiple sclerosis.
32. Sugar can cause hemorrhoids.
33. Sugar can cause varicose veins.
34. Sugar can elevate glucose and insulin responses in oral contraceptive users.
35. Sugar can lead to periodontal disease.
36. Sugar can contribute to osteoporosis.
37. Sugar contributes to saliva acidity.
38. Sugar can cause a decrease in insulin sensitivity.
39. Sugar can lower the amount of Vitamin E in the blood.
40. Sugar can decrease growth hormone.
41. Sugar can increase cholesterol.
42. Sugar can increase the systolic blood pressure.
43. Sugar can cause drowsiness and decreased activity in children.
44. High sugar intake increases advanced glycation end products (AGEs) (Sugar bound non- enzymatically to protein)
45. Sugar can interfere with the absorption of protein.
46. Sugar causes food allergies.
47. Sugar can contribute to diabetes.
48. Sugar can cause toxemia during pregnancy.
49. Sugar can contribute to eczema in children.
50. Sugar can cause cardiovascular disease.
51. Sugar can impair the structure of DNA
52. Sugar can change the structure of protein.
53. Sugar can make our skin age by changing the structure of collagen.
54. Sugar can cause cataracts.
55. Sugar can cause emphysema.
56. Sugar can cause atherosclerosis.
57. Sugar can promote an elevation of low density lipoproteins (LDL).
58. High sugar intake can impair the physiological homeostasis of many systems in the body.
59. Sugar lowers the enzymes ability to function.
60. Sugar intake is higher in people with Parkinson’s disease.
61. Sugar can cause a permanent altering of the way the proteins act in the body.
62. Sugar can increase the size of the liver by making the liver cells divide.
63. Sugar can increase the amount of liver fat.
64. Sugar can increase kidney size and produce pathological changes in the kidney.
65. Sugar can damage the pancreas.
66. Sugar can increase the body’s fluid retention.
67. Sugar is enemy #1 of the bowel movement.
68. Sugar can cause myopia (nearsightedness).
69. Sugar can compromise the lining of the capillaries.
70. Sugar can make the tendons more brittle.
71. Sugar can cause headaches, including migraine.
72. Sugar plays a role in pancreatic cancer in women.
73. Sugar can adversely affect school children’s grades and cause learning disorders.
74. Sugar can cause an increase in delta, alpha and theta brain waves.
75. Sugar can cause depression.
76. Sugar increases the risk of gastric cancer.
77. Sugar can cause dyspepsia (indigestion).
78. Sugar can increase your risk of getting gout.
79. Sugar can increase the levels of glucose in an oral glucose tolerance test over the ingestion of complex carbohydrates.
80. Sugar can increase the insulin responses in humans consuming high-sugar diets compared to low-sugar diets.
81. High refined sugar diet reduces learning capacity.
82. Sugar can cause less effective functioning of two blood proteins, albumin, and lipoproteins, which may reduce the body’s ability to handle fat and cholesterol.
83. Sugar can contribute to Alzheimer’s disease.
84. Sugar can cause platelet adhesiveness.
85. Sugar can cause hormonal imbalance; some hormones become  underactive and others become overactive.
86. Sugar can lead to the formation of kidney stones.
87. Sugar can lead to the hypothalamus to becoming highly sensitive to a large variety of stimuli.
88. Sugar can lead to dizziness.
89. Diets high in sugar can cause free radicals and oxidative stress.
90. High sucrose diets of subjects with peripheral vascular disease significantly increases platelet adhesion.
91. High sugar diet can lead to biliary tract cancer.
92. Sugar feeds cancer.
93. High sugar consumption by pregnant adolescents is associated with a twofold increased risk for delivering a small-for-gestational-age (SGA) infant.
94. High sugar consumption can lead to a substantial decrease in gestation duration among adolescents.
95. Sugar slows food’s travel time through the gastrointestinal tract.
96. Sugar increases the concentration of bile acids in stools and bacterial enzymes in the colon. This can modify bile to produce cancer-causing compounds and colon cancer.
97. Sugar increases estradiol (the most potent form of naturally occurring estrogen) in men.
98. Sugar combines and destroys phosphatase, an enzyme which makes the process of digestion more difficult.
99. Sugar can be a risk factor of gallbladder cancer.
100. Sugar is an addictive substance.
101. Sugar can be intoxicating, similar to alcohol.
102. Sugar can exacerbate PMS.
103. Sugar given to premature babies can affect the amount of carbon dioxide they produce.
104. Decrease in sugar intake can increase emotional stability.
105. The body changes sugar into 2 to 5 times more fat in the bloodstream than it does starch.
106. The rapid absorption of sugar promotes excessive food intake in obese subjects.
107. Sugar can worsen the symptoms of children with attention deficit hyperactivity disorder (ADHD).
108. Sugar adversely affects urinary electrolyte composition.
109. Sugar can slow down the ability of the adrenal glands to function.
110. Sugar has the potential to induce abnormal metabolic processes in a normal healthy individual and to promote chronic degenerative diseases.
111. I.V.s (intravenous feedings) of sugar water can cut off oxygen to the brain.
112. High sucrose intake could be an important risk factor in lung cancer.
113. Sugar increases the risk of polio.
114. High sugar intake can cause epileptic seizures.
115. Sugar causes high blood pressure in obese people.
116. In Intensive Care Units: Limiting sugar saves lives.
117. Sugar may induce cell death.
118. Sugar may impair the physiological homeostasis of many systems in living organisms.
119. In juvenile rehabilitation camps, when children were put on a low sugar diet, there was a 44% drop in antisocial behavior.
120. Sugar can cause gastric cancer.
121. Sugar dehydrates newborns.
122. Sugar can cause gum disease.
123. Sugar increases the estradiol in young men.
124. Sugar can cause low birth-weight babies.

——————————————————————————–

SOURCES

1. Sanchez, A., et al. “Role of Sugars in Human Neutrophilic Phagocytosis,” American Journal of Clinical Nutrition.
Nov 1973;261:1180_1184. Bernstein, J., et al. “Depression of Lymphocyte Transformation Following Oral Glucose
Ingestion.” American Journal of Clinical Nutrition.1997;30:613.

2. Couzy, F., et al.”Nutritional Implications of the Interaction Minerals,” Progressive Food and Nutrition Science 17;1933:65-87.

3. Goldman, J., et al. “Behavioral Effects of Sucrose on Preschool Children.” Journal of Abnormal Child Psychology.1986;
14(4):565_577.

4. Scanto, S. and Yudkin, J. “The Effect of Dietary Sucrose on Blood Lipids, Serum Insulin, Platelet Adhesiveness and
Body Weight in Human Volunteers,” Postgraduate Medicine Journal. 1969;45:602_607.

5. Ringsdorf, W., Cheraskin, E. and Ramsay R. “Sucrose,Neutrophilic Phagocytosis and Resistance to Disease,”
Dental Survey. 1976;52(12):46_48.

6. Cerami, A., Vlassara, H., and Brownlee, M.”Glucose and Aging.” Scientific American. May 1987:90.

Lee, A. T. and Cerami, A. “The Role of Glycation in Aging.” Annals of the New York Academy of Science; 663:63-67.

7. Albrink, M. and Ullrich I. H. “Interaction of Dietary Sucrose and Fiber on Serum Lipids in Healthy Young Men Fed High
Carbohydrate Diets.” American Journal of Clinical Nutrition. 1986;43:419-428.

Pamplona, R., et al. “Mechanisms of Glycation in Atherogenesis.” Med Hypotheses. Mar 1993;40(3):174-81.

8. Kozlovsky, A., et al. “Effects of Diets High in Simple Sugars on Urinary Chromium Losses.” Metabolism.
June 1986;35:515_518.

9. Takahashi, E., Tohoku University School of Medicine, Wholistic Health Digest. October 1982:41.

10. Kelsay, J., et al. “Diets High in Glucose or Sucrose and Young Women.” American Journal of Clinical Nutrition.
1974;27:926_936.

Thomas, B. J., et al. “Relation of Habitual Diet to Fasting Plasma Insulin Concentration and the Insulin Response to
Oral Glucose,” Human Nutrition Clinical Nutrition. 1983; 36C(1):49_51.

11. Fields, M.., et al. “Effect of Copper Deficiency on Metabolism and Mortality in Rats Fed Sucrose or Starch Diets,”
Journal of Clinical Nutrition. 1983;113:1335_1345.

12. Lemann, J. “Evidence that Glucose Ingestion Inhibits Net Renal Tubular Reabsorption of Calcium and Magnesium.”
Journal Of Clinical Nutrition. 1976 ;70:236_245.

13. Acta Ophthalmologica Scandinavica. Mar 2002;48;25.

Taub, H. Ed. “Sugar Weakens Eyesight,” VM NEWSLETTER;May 1986:6

14. “Sugar, White Flour Withdrawal Produces Chemical Response.” The Addiction Letter .Jul 1992:4.

15. Dufty, William. Sugar Blues. (New York:Warner Books, 1975).

16. Ibid.

17. Jones, T. W., et al. “Enhanced Adrenomedullary Response and Increased Susceptibility to Neuroglygopenia:
Mechanisms Underlying the Adverse Effect of Sugar Ingestion in Children.” Journal of Pediatrics. Feb 1995;126:171-7.

18. Ibid.

19. Lee, A. T.and Cerami A. “The Role of Glycation in Aging.” Annals of the New York Academy of Science.1992;663:63-70.

20. Abrahamson, E. and Peget, A.. Body, Mind and Sugar. (New York:Avon,1977.}

21. Glinsmann, W., Irausquin, H., and Youngmee, K. “Evaluation of Health Aspects of Sugar Contained in Carbohydrate
Sweeteners. F. D. A. Report of Sugars Task Force.” 1986:39.

Makinen K.K.,et al. “A Descriptive Report of the Effects of a 16_month Xylitol Chewing_gum Programme Subsequent
to a 40_month Sucrose Gum Programme.” Caries Research. 1998; 32(2)107_12.

22. Keen, H., et al. “Nutrient Intake, Adiposity, and Diabetes.” British Medical Journal. 1989; 1: 655_658

23. Persson P. G., Ahlbom, A., and Hellers, G. Epidemiology. 1992;3:47-52.

24. Yudkin, J. New York: Sweet and Dangerous.:Bantam Books:1974: 129.

25. Darlington, L., Ramsey, N. W. and Mansfield, J. R. “Placebo_Controlled, Blind Study of Dietary Manipulation
Therapy in Rheumatoid Arthritis,” Lancet. Feb 1986;8475(1):236_238.

26. Powers, L. “Sensitivity: You React to What You Eat.” Los Angeles Times. (Feb. 12, 1985).

Cheng, J., et al. “Preliminary Clinical Study on the Correlation Between Allergic Rhinitis and Food Factors.”
Lin Chuang Er Bi Yan Hou Ke Za Zhi Aug 2002;16(8):393-396.

27. Crook, W. J. The Yeast Connection. (TN:Professional Books, 1984)..

28. Heaton, K. “The Sweet Road to Gallstones.” British Medical Journal. Apr 14, 1984; 288: 1103_1104.

Misciagna, G., et al. American Journal of Clinical Nutrition. 1999;69:120-126.

29. Yudkin, J. “Sugar Consumption and Myocardial Infarction.” Lancet..Feb 6, 1971:1(7693):296-297.

Suadicani, P., et al. “Adverse Effects of Risk of Ishaemic Heart Disease of Adding Sugar to Hot Beverages in
Hypertensives Using Diuretics.” Blood Pressure. Mar 1996;5(2):91-71.

30. Cleave, T. The Saccharine Disease. (New Canaan, CT: Keats Publishing, 1974).

31. Erlander, S. “The Cause and Cure of Multiple Sclerosis, “The Disease to End Disease.” Mar 3, 1979;1(3):59_63.

32. Cleave, T. The Saccharine Disease. (New Canaan, CT: Keats Publishing, 1974.)

33. Cleave, T. and Campbell, G. (Bristol, England:Diabetes, Coronary Thrombosis and the Saccharine Disease:
John Wrightand Sons, 1960).

34. Behall, K. “Influ ence of Estrogen Content of Oral Contraceptives and Consumption of Sucrose on Blood Parameters.”
Disease Abstracts International. 1982;431437.

35. Glinsmann, W., Irausquin, H., and K. Youngmee. Evaluation of Health Aspects of Sugar Contained in Carbohydrate
Sweeteners. F. D. A. Report of Sugars Task Force.1986;39:36_38.

36. Tjäderhane, L. and Larmas, M. “A High Sucrose Diet Decreases the Mechanical Strength of Bones in Growing Rats.”
Journal of Nutrition. 1998:128:1807_1810.

37. Appleton, N. New York: Healthy Bones. Avery Penguin Putnam:1989.

38. Beck_Nielsen H., Pedersen O., and Schwartz S. “Effects of Diet on the Cellular Insulin Binding and the Insulin
Sensitivity in Young Healthy Subjects.” Diabetes. 1978;15:289_296 .

39. Journal of Clinical Endocrinology and Metabolism. Aug 2000.

40. Gardner, L. and Reiser, S. “Effects of Dietary Carbohydrate on Fasting Levels of Human Growth Hormone and
Cortisol.” Proceedings of the Society for Experimental Biology and Medicine. 1982;169:36_40.

41. Reiser, S. “Effects of Dietary Sugars on Metabolic Risk Factors Associated with Heart Disease.” Nutritional Health.
1985;203_216.

42. Hodges, R., and Rebello, T. “Carbohydrates and Blood Pressure.” Annals of Internal Medicine. 1983:98:838_841.

43. Behar, D., et al. “Sugar Challenge Testing with Children Considered Behaviorally Sugar Reactive.” Nutritional Behavior.
1984;1:277_288.

44. Furth, A. and Harding, J. “Why Sugar Is Bad For You.” “New Scientist.” Sep 23, 1989;44.

45. Simmons, J. “Is The Sand of Time Sugar?” LONGEVITY. June 1990: 49_53.

46. Randolph, T. G. et al. “Allergic Reactions Following Intravenous Injection of Corn Sugar (Dextrose)”.
Archives of Surgery. 1950;64:554-564.

47. “Sucrose Induces Diabetes in Cat.” Federal Protocol. 1974;6(97).

48. Cleave, T.:The Saccharine Disease: (New Canaan Ct: Keats Publishing, Inc., 1974).131.

49. Ibid. 132.

50. Vaccaro O., Ruth, K. J. and Stamler J. “Relationship of Postload Plasma Glucose to Mortality with 19_yr Follow_up.”
Diabetes Care. Oct 15,1992;10:328_334.

Tominaga, M., et al, “Impaired Glucose Tolerance Is a Risk Factor for Cardiovascular Disease, but Not Fasting Glucose.”
Diabetes Care. 1999:2(6):920-924.

51. Lee, A. T. and Cerami, A. “Modifications of Proteins and Nucleic Acids by Reducing Sugars: Possible Role in Aging.”
Handbook of the Biology of Aging. ( New York: Academic Press, 1990.).

52. Monnier, V. M. “Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process.” Journal of Gerontology
1990:45(4 ):105_110.

53. Dyer, D. G., et al. “Accumulation of Maillard Reaction Products in Skin Collagen in Diabetes and Aging.” Journal of
Clinical Investigation. 1993:93(6):421_22.

54. Veromann, S.et al.”Dietary Sugar and Salt Represent Real Risk Factors for Cataract Development.” Ophthalmologica.
2003 Jul-Aug;217(4):302-307.

55. Monnier, V. M. “Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process.” Journal of Gerontology.
1990:45(4):105_110.

56. Pamplona, R., et al. “Mechanisms of Glycation in Atherogenesis.” Medical Hypotheses . 1990: 174_181.

57. Lewis, G. F. and Steiner, G. “Acute Effects of Insulin in the Control of Vldl Production in Humans. Implications for
Theinsulin-resistant State.” Diabetes Care. 1996 Apr;19(4):390-3 R. Pamplona, M. .J., et al. “Mechanisms of Glycation
in Atherogenesis.” Medical Hypotheses. 1990;40:174-181.

58. Ceriello, A. “Oxidative Stress and Glycemic Regulation.” Metabolism. Feb 2000;49(2 Suppl 1):27-29.

59. Appleton, Nancy. New York; Lick the Sugar Habit. Avery Penguin Putnam, 1988.

60. Hellenbrand, W. “Diet and Parkinson’s Disease. A Possible Role for the Past Intake of Specific Nutrients. Results
from a Self-administered Food-frequency Questionnaire in a Case-control Study.” Neurology. Sep 1996;47(3):644-650.

61. Cerami, A., Vlassara, H., and Brownlee, M. “Glucose and Aging.” Scientific American. May 1987: 90.

62. Goulart, F. S. “Are You Sugar Smart?” American Fitness. March_April 1991: 34_38.

63. Ibid.

64. Yudkin, J., Kang, S. and Bruckdorfer, K. “Effects of High Dietary Sugar.” British Journal of Medicine. Nov 22, 1980;1396.

65. Goulart, F. S. “Are You Sugar Smart?” American Fitness. March_April 1991: 34_38. Milwakuee, WI,: damage pancreas

66. Ibid. fluid retention

67. Ibid. bowel movement

68. Ibid. nearsightedness

69. Ibid. compromise the lining of the capillaries

70. Nash, J. “Health Contenders.” Essence. Jan 1992; 23: 79_81.

71. Grand, E. “Food Allergies and Migraine.”Lancet. 1979:1:955_959.

72. Michaud, D. “Dietary Sugar, Glycemic Load, and Pancreatic Cancer Risk in a Prospective Study.” J Natl Cancer Inst.
Sep 4, 2002 ;94(17):1293-300.

73. Schauss, A. Diet, Crime and Delinquency. (Berkley Ca; Parker House, 1981.)

74. Christensen, L. “The Role of Caffeine and Sugar in Depression.” Nutrition Report. Mar 1991;9(3):17-24.

75. Ibid.

76. Cornee, J., et al. “A Case-control Study of Gastric Cancer and Nutritional Factors in Marseille, France,”
European Journal of Epidemiology. 1995;11:55-65.

77. Yudkin, J. Sweet and Dangerous.(New York:Bantam Books,1974) 129.

78. Ibid, 44

79. Reiser, S., et al. “Effects of Sugars on Indices on Glucose Tolerance in Humans.” American Journal of Clinical
Nutrition. 1986:43;151-159.

80. Reiser,S., et al. “Effects of Sugars on Indices on Glucose Tolerance in Humans.” American Journal of Clinical Nutrition.
1986;43:151-159.

81. Molteni, R, et al. “A High-fat, Refined Sugar Diet Reduces Hippocampal Brain-derived Neurotrophic Factor,
Neuronal Plasticity, and Learning.” NeuroScience. 2002;112(4):803-814.

82. Monnier, V., “Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process.” Journal of Gerontology.
1990;45:105-111.

83. Frey, J. “Is There Sugar in the Alzheimer’s Disease?” Annales De Biologie Clinique. 2001; 59 (3):253-257.

84. Yudkin, J. “Metabolic Changes Induced by Sugar in Relation to Coronary Heart Disease and Diabetes.”
Nutrition and Health. 1987;5(1-2):5-8.

85. Ibid.

86. Blacklock, N. J., “Sucrose and Idiopathic Renal Stone.” Nutrition and Health. 1987;5(1-2):9-

Curhan, G., et al. “Beverage Use and Risk for Kidney Stones in Women.” Annals of Internal Medicine. 1998:28:534-340.

87. Journal of Advanced Medicine. 1994;7(1):51-58.

88. Ibid

89. Ceriello, A. “Oxidative Stress and Glycemic Regulation.” Metabolism. Feb 2000;49(2 Suppl 1):27-29.

90. Postgraduate Medicine.Sept 1969:45:602-07.

91. Moerman, C. J., et al. “Dietary Sugar Intake in the Etiology of Biliary Tract Cancer.” International Journal of
Epidemiology . Ap 1993;.2(2):207-214.

92. Quillin, Patrick, “Cancer’s Sweet Tooth,” Nutrition Science News. Ap 2000.

Rothkopf, M.. Nutrition. July/Aug 1990;6(4).

93. Lenders, C. M. “Gestational Age and Infant Size at Birth Are Associated with Dietary Intake among Pregnant
Adolescents.” Journal of Nutrition. Jun 1997;1113- 1117.

94. Ibid.

95. Bostick, R. M., et al. “Sugar, Meat.and Fat Intake and Non-dietary Risk Factors for Colon Cancer Incidence in
Iowa Women.” Cancer Causes & Control. 1994:5 :38-53.

96. Ibid.

Kruis, W., et al. “Effects of Diets Low and High in Refined Sugars on Gut Transit, Bile Acid Metabolism and Bacterial
Fermentation.” Gut. 1991;32:367-370.

Ludwig, D. S., et al. “High Glycemic Index Foods, Overeating, And Obesity.” Pediatrics. Mar 1999;103(3):26-32.

97. Yudkin, J and Eisa, O. “Dietary Sucrose and Oestradiol Concentration in Young Men”.

Annals of Nutrition and Metabolism. 1988:32(2):53-55.

98. Lee, A. T. and Cerami A. “The Role of Glycation in Aging.” Annals of the New York Academy of Science.
1992; 663:63-70.

99. Moerman, C., et al.”Dietary Sugar Intake in the Etiology of Biliary Tract Cancer.” International Journal of
Epidemiology. Ap 1993; 22(2):207-214.

100. “Sugar, White Flour Withdrawal Produces Chemical Response.” The Addiction Letter. Jul 1992:4.

Colantuoni, C., et al. “Evidence That Intermittent, Excessive Sugar Intake Causes Endogenous Opioid Dependence.”
Obes Res. Jun 2002 ;10(6):478-488.

Annual Meeting of the American Psychological Society, Toronto, June 17, 2001.

Www.mercola,com/2001/june/30/sugar.htm

101. Ibid.

102. The Edell Health Letter. Sept 1991;7:1.

103. Sunehag, A. L., et al. “Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition”
Diabetes. 1999 ;48 7991_800.

104. Christensen L., et al. “Impact of A Dietary Change on Emotional Distress.” Journal of Abnormal Psychology .
1985;94(4):565_79.

105. Nutrition Health Review. Fall 85. changes sugar into fat faster than fat

106. Ludwig, D. S., et al. “High Glycemic Index Foods, Overeating and Obesity.” Pediatrics.  March 1999;103(3):26-32.

107. Pediatrics Research. 1995;38(4):539-542.

Berdonces, J. L. “Attention Deficit and Infantile Hyperactivity.” Rev Enferm. Jan 2001;4(1)11-4

108. Blacklock, N. J. “Sucrose and Idiopathic Renal Stone.” Nutrition Health. 1987;5(1 & 2):9-17.

109. Lechin, F., et al. “Effects of an Oral Glucose Load on Plasma Neurotransmitters in Humans.” Neurophychobiology.
1992;26(1-2):4-11.

110. Fields, M. Journal of the American College of Nutrition. Aug 1998;17(4):317_321.

111. Arieff, A. I. Veterans Administration Medical Center in San Francisco. San Jose Mercury; June 12/86. IVs of
sugar water can cut off oxygen to the brain.

112. De Stefani, E.”Dietary Sugar and Lung Cancer: a Case_control Study in Uruguay.” Nutrition and Cancer.
1998;31(2):132_7.

113. Sandler, Benjamin P. Diet Prevents Polio. Milwakuee, WI,:The Lee Foundation for for Nutritional Research, 1951.

114. Murphy, Patricia. “The Role of Sugar in Epileptic Seizures.” Townsend Letter for Doctors and Patients.
May, 2001. Murphy Is Editor of Epilepsy Wellness Newsletter, 1462 West 5th Ave., Eugene, Oregon 97402

115. Stern, N. & Tuck, M. “Pathogenesis of Hypertension in Diabetes Mellitus.” Diabetes Mellitus, a Fundamental
and Clinical Test. 2nd Edition, (PhiladelphiA; A:Lippincott Williams & Wilkins, 2000)943-957.

116. Christansen, D. “Critical Care: Sugar Limit Saves Lives.” Science News. June 30, 2001; 159:404.

117. Donnini, D. et al. “Glucose May Induce Cell Death through a Free Radical-mediated Mechanism.”Biochem
Biohhys Res Commun. Feb 15, 1996:219(2):412-417.

118. Ceriello, A. “Oxicative Stress and Glycemic Regulation.” Metabolism. Feb 2000;49(Suppl I):27-29.

119. Schoenthaler, S. The Los Angeles Probation Department Diet-Behavior Program: Am Empirical Analysis of Six
Institutional Settings. Int J Biosocial Res 5(2):88-89.

120. Cornee, J., et al. “A Case-control Study of Gastric Cancer and Nutritional Factors in Marseille, France.” European
Journal of Epidemiology 11 (1995):55-65.

121. “Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition. Diabetes.
1999 Apr;48(4):791-800.

122. Glinsmann, W., et al. “Evaluation of Health Aspects of Sugar Contained in Carbohydrate

123. Sweeteners.” FDA Report of Sugars Task Force (1986) 39. 123. Yudkin, J. and Eisa, O. “Dietary Sucrose and
Oestradiol Concentration in Young Men. Annals of Nutrition and Metabolism. 1988;32(2):53-5.

124. Lenders, C. M. “Gestational Age and Infant Size at Birth Are Associated with Dietary Intake Among Pregnant
Adolescents.” Journal of Nutrition 128.1998:1807-1810